L-Tryptophan, Sensory Issues, and the Three Pathways that affect Serotonin

Alcoholism - Why People Turn to It and How It Destroys the Brain

Many people, especially autistic people, or people with bipolar disorder, or ADHD, have a neurological baseline where glutamate (the brain's "go" signal) runs high and GABA (the "brake") runs low. Alcohol temporarily patches that imbalance, which is why vulnerable populations are drawn to it at disproportionate rates.   The catch: alcohol is a neurotoxin. When it wears off, GABA crashes even lower, glutamate spikes even higher, and every symptom gets worse. Repeat this enough and you're looking at hippocampal atrophy, suppressed neurogenesis, blunted reward circuits, and memories that simply never encode, which is why being around heavy drinkers can feel like being gaslit.   Addiction isn't a willpower problem. It's a nervous system trying to fill a real neurochemical void. Movement, nature, and other GABA-supporting habits can build new neural pathways over time, but the alternative has to come before the substance is taken away.

The Problems With Spanking Children, Corporal Punishment, Shame Avoidance

What this episode covers: This episode explains the systemic effects of spanking - not just that it's harmful, but how it creates lasting patterns. I'll show you: * What happens in the nervous system when a child is spanked * The dysfunctional beliefs children develop from these experiences * How shame avoidance perpetuates the pattern across generations * Why protective behaviors (formed in response to spanking) show up differently across contexts but stem from the same root cause * The pathway to breaking the cycle Key statistics: * 94% of 3-4 year olds in the US are spanked * Spanking in childhood is associated with moderate to heavy drinking, drug use, and suicide attempts in adulthood (even after controlling for other forms of abuse) Sources: * Fragile Families study on spanking prevalence [https://pmc.ncbi.nlm.nih.gov/articles/PMC3114638/] * Long-term mental health outcomes [https://www.sciencedirect.com/science/article/abs/pii/S0145213423000030]

How Glutamate EAAT2 Enzyme Affects Autism - I am NOT Separate from Autism

Who I am - my personality - are not separate.  I share a short anecdote about how I'm not separate from being autistic, and share the HUGE news released in 2025 that impacts all autistics every day.   I discuss the neuroscience behind how autism involves increased glutamate from reduced activity in an enzyme called EAAT2, reduced GABA, reduced GABA sensitivity, and reduced vitamin D, this study here: https://www.frontiersin.org/journals/psychiatry/articles/10.3389/fpsyt.2025.1562631/full

Autistic Oxytocin Systems Compared to Allistic Oxytocin Systems

In this episode, I unpack my suspicions about how the autistic brain might produce oxytocin differently from the allistic brain, and what that could mean neurologically and behaviorally.  Connect with us with PATTERNS not with PERFORMANCE.

L-Tryptophan, Sensory Issues, and the Three Pathways that affect Serotonin

Additional Note, discovered later on: Active B6 (aka P5P) is a crucial co-factor for L-Tryptophan -> Serotonin conversion, so ensure this cofactor is met for optimal conversion. This episode explores how I found solutions to my lifetime of sensory issues which I accepted as normal. L-Tryptophan allowed me to create enough serotonin in a rate-limited lower risk way to be able to nearly resolve sensory issues & might have strong potential to benefit the autistic brain.